Artificial Life in the Fight Against Cancer
نویسنده
چکیده
Blood vessel growth is a fascinating example of an adaptive, morphologically plastic network formation process driven by complex interactions between individual cells in the vessel and between the cells and their dynamic extracellular environment. Under normal conditions this can generate a well-adapted hierarchical branching structure. However in tumours, blood vessels become maladapted, leaky and bulbous, resulting in increased hypoxia and tumour cell metastasis. A method to switch tumour blood vessels back to a normal network could reduce metastasis and thus represents a significant goal in cancer therapy. However, studying human disease and the abnormalities that lead to pathological phenotypes is a monumentally difficult task. Humans don’t tend to offer themselves for experimentation and animal disease models can be hard to establish. Probing the inner workings of in vivo systems present numerous technical challenges, though boundaries continue to be broken. Further, the normal fundamental mechanisms controlling development are of course not fully understood, let alone their perturbation by environmental changes in disease.
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